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Toxic and Nutritional Optic Neuropathies Simultaneous impairment of vision in the two eyes, with central or centrocecal scotomas, is caused not by a demyelinative process but usually by a toxic or nutritional process The condition is observed most often in the chronically alcoholic, malnourished patient Impairment of visual acuity evolves over several days or a week or two, and examination discloses bilateral, roughly symmetrical central or centrocecal scotomas, the peripheral elds being intact With appropriate treatment (nutritious diet and B vitamins) instituted soon after the onset of amblyopia, complete recovery is possible; if treatment is delayed, patients are left with varying degrees of permanent defect in central vision and pallor of the temporal portions of the optic discs This disorder has commonly been referred to as tobacco-alcohol amblyopia, the implication being that it is due to the toxic effects of tobacco or alcohol, or both.

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These clinical phenomena are referable to any part of the CNS but tend to be stereotyped in an individual patient The most common phenomena are dysarthria and ataxia, paroxysmal pain and dysesthesia in a limb, ashing lights, paroxysmal itching, or tonic seizures, taking the form of exion (dystonic) spasm of the hand, wrist, and elbow with extension of the lower limb The paroxysmal symptoms, particularly the tonic spasms, may be triggered by sensory stimuli or can be elicited by hyperventilation On a few occasions we have seen dystonic hand and arm spasms as the rst symptoms; an acute plaque was detected in the opposite internal capsule In advanced cases, the spasms may involve all four limbs and even a degree of opisthotonos The cause of paroxysmal phenomena is uncertain They have been attributed by Halliday and McDonald to ephaptic transmission ( cross-talk ) between adjacent demyelinated axons within a lesion.

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In fact, the disorder is one of nutritional de ciency and is more properly desig-.

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AC Adapter A type of power supply that converts AC power to voltages needed for the PC system. AC adapters are usually used in portable PC systems. Access Methods Also known as network access, these are the methods by which a device communicates on a network. Network access provides a standard that all devices that wish to communicate on a network must abide by in order to eliminate communication conflicts. Active Matrix Display Based on thin-film transistor (TFT) technology. Instead of having two rows of transistors, active matrix displays have a transistor at every pixel, which enables much quicker display changes than passive matrix displays and produces display quality comparable to a CRT. Adapter Card Refers to any PC that is added to the motherboard to enhance functionality or expansion. Monitor cards and network cards are examples of adapter cards. AGP Accelerated graphics port. A monitor interface designed for highperformance graphics processing. AMR Audio Modem Riser. A card that allows for connection of multimedia and communications to a PC. AMR technology utilizes excess capacity in the CPU to emulate hardware devices. ANSI.SYS A DOS system file that is loaded by CONFIG.SYS if required. This file loads an extended character set for use by DOS and DOS applications that includes basic drawing and color capabilities. Normally used for drawing and filling different boxes for menu systems, it is seldom used today. By default, it carries no attributes and is not required for OS startup. Antenna Radiates and receives radio frequency signals. Usually used with wireless technology. AppleTalk networking.

An Activity is an Android application. Activities are covered in more depth in the following chapters.

Carbamazepine is usually effective in controlling such attacks These transitory symptoms appear suddenly, may recur frequently for several days or weeks, sometimes longer, and then remit completely, ie, they exhibit the temporal pro le of a relapse or an exacerbation It is sometimes dif cult to determine whether they represent an exacerbation or a new lesion Years ago, Thygessen pointed out, in an analysis of 105 exacerbations in 60 patients, that there were new symptoms in only 19 percent; in the remainder there was only a recurrence of old symptoms Another problem is that the original lesion may have been asymptomatic This is most obviously re ected in the many patients who are found to have impaired visual evoked responses but have never had symptomatic visual changes Thus, new symptoms and signs may be manifestations of previously formed but asymptomatic plaques.

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